Opa1 Neuron

Neuron induction medium NIM promoted OPA1 RGC generation detected

OPA1 Mutations Associated with PD Cause an Early Loss of DA Synapses

OPA1 mRNA and protein expression in spiral ganglion neurons of the rat

iPSCderived dopaminergic neurons from syndromic OPA1 parkinsonism

Principle of mitochondrial quality control with focus on OPA1 function

OPA1 mRNA and protein expression in spiral ganglion neurons of the rat

Mitochondrial Membrane DynamicsFunctional Positioning of OPA1

Opa1 promotes neuronal survival and rescues mitochondrial morphology

OPA1 expression in the normal rat retina and optic nerve Abstract

Caspase inhibitors and protein synthesis inhibitors diminish OPA1

Nterminal cleavage of the mitochondrial fusion GTPase OPA1 occurs via

OPA1 Deficiency Impairs NGF Signaling and Drives Sympathetic

Interface 7 of the OPA1 assembly is essential for the regulation of

Mitochondrial Membrane DynamicsFunctional Positioning of OPA1

Caspase inhibitors and protein synthesis inhibitors diminish OPA1

Changes in neuronal mitochondrial networks and OPA1 protein cleavage

OPA1 protein expression in the vestibular organ of the rat a b OPA1

OPA1 mRNA and protein expression in spiral ganglion neurons of the rat

OPA1 mutation reduces OPA1 protein expression and induces apoptotic

OPA1 mutation triggers activation of NMDA receptors in Opa1enu mice

OPA1 promotes ferroptosis by augmenting mitochondrial ROS and

Loss of OPA1 triggers apoptosis a kinetic of appearance of the

Single double and triple Opa1 Cterminal Cys mutants generate the 180

Changes in neuronal mitochondrial networks and OPA1 protein cleavage

Loss of OPA1 Perturbates the Mitochondrial Inner Membrane Structure and

OPA1 mutation reduces OPA1 protein expression and induces apoptotic

Mitochondrial Membrane DynamicsFunctional Positioning of OPA1

Opa1 and cytochrome c release following global brain ischemia Opa1 and

Loss of OPA1 induces mitochondrial membrane potential reduction m was

Loss of OPA1 triggers apoptosis a kinetic of appearance of the

OPA1 protein expression in the vestibular organ of the rat a b OPA1

Frontiers Optimized OPA1 Isoforms 1 and 7 Provide Therapeutic Benefit

Nterminal cleavage of the mitochondrial fusion GTPase OPA1 occurs via

Proapoptotic stimuli induce a unique Nterminal cleavage of OPA1 in

Opa1 functions downstream of calpain activation A neurons were

OPA1 participates in regulation of mitochondrial network morphology in

OPA1 in MC4R Neurons Regulates Dietary Fat Intake and Body Weight in

Opa1 mice exhibit reduced Opa1 immunofluorescence in the retinal

Expression of Nterminal Opa1 mutants has no effect on mitochondrial

Colocalizations of the signals of Drp1 or Opa1 with NeuNpositive

OPA1 和致病突变体扰乱线粒体类核分布,Cell Death & Disease - X-MOL

OPA1 Cell Death Disease XMOL

Autophagy controls the pathogenicity of OPA1 mutations in dominant

Schematic of OPA1 processing Download Scientific Diagram

OPA1 Deficiency Impairs NGF Signaling and Drives Sympathetic

Changes in neuronal mitochondrial networks and OPA1 protein cleavage

OPA1 expression in the normal rat retina and optic nerve Abstract

OPA1 Controls Apoptotic Cristae Remodeling Independently from

The Balance of MFN2 and OPA1 in Mitochondrial Dynamics Cellular

Contrasting pathophysiological mechanisms of OPA1 mutations in

Frontiers Mitochondrial OMA1 and OPA1 as Gatekeepers of Organellar

Targeting the OPA1 pathway in Autosomal Dominant Optic Atrophy ADOA

The short variant of the mitochondrial dynamin OPA1 maintains

Blockade of glutamate receptor activation blocks OPA1 release in

Drosophila model to clarify the pathological significance of OPA1 in

OPA1 Antibody PA557874

Alteration of total OPA1 expression and distribution in the retinas of

Box plot showing the OPA1 protein content relative to GAPDH protein in

Opantimirs A class of antagonizing microRNAs that upregulate Opa1 and

Schematic Representation of the Hierarchic Features Rescued by OPA1 and

OPA1 Expression in the Normal Rat Retina and Optic Nerve PMC

Morphological analysis of mitochondria in Opa1 conditional knockout

Opa1 and Fis1 protein levels are increased in neural retinas of cbs

Opa1 Overexpression Ameliorates the Phenotype of Two Mitochondrial

OPA1 and Angiogenesis Beyond the Fusion Function Cell Metabolism

OPA1 mRNA and protein expression in the rat cochlea ac OPA1 mRNA

OPA1 regulation of mitochondrial dynamics in skeletal and cardiac

Derivation and Characterization of Isogenic OPA1 Mutant and Control

Overall schematic for the involving of OPA1 in mitochondrial

Proteolytic Cleavage of Opa1 Stimulates Mitochondrial Inner Membrane

OPA1 as a Cancer Target Molecular Mechanisms Structural Insights and

Schematic Representation of the Hierarchic Features Rescued by OPA1 and

OPA1 Antibody optic atrophy protein 1 Rabbit IgG Antibody

Figure 1 from OPA1 loss of function affects in vitro neuronal

Proteolytic Processing of OPA1 Links Mitochondrial Dysfunction to

OPA1 in MC4R Neurons Regulates Dietary Fat Intake and Body Weight in

OPA1 drives macrophage metabolism and functional commitment via p65

Derivation and Characterization of Isogenic OPA1 Mutant and Control

OPA1 promotes ferroptosis by augmenting mitochondrial ROS and

OMA1 degrades OPA1 in AFG3L1AFG3L2deficient MEFs A Stability of

Loss of OPA1 triggers apoptosis a kinetic of appearance of the

OPA1 Controls Apoptotic Cristae Remodeling Independently from

OPA1 and PARL Keep a Lid on Apoptosis Cell

Proteolytic Cleavage of Opa1 Stimulates Mitochondrial Inner Membrane

Reconstitution of the Human Nigrostriatal Pathway onaChip Reveals

Mitochondrial Function OMA1 and OPA1 the Grandmasters of

Optic Atrophy 1 Controls Human Neuronal Development by Preventing

Developmental and Tumor Angiogenesis Requires the MitochondriaShaping

Reconstitution of the Human Nigrostriatal Pathway onaChip Reveals

Optic Atrophy 1 Controls Human Neuronal Development by Preventing

Overexpression of S1OPA1 increased neuronal degeneration and

Increased optic atrophy type 1 OPA1 expression blocks mitochondrial

Mitochondrial Optic Atrophy OPA 1 Processing Is Altered in Response

Optic Atrophy 1 Controls Human Neuronal Development by Preventing

Prohibitin levels regulate OMA1 activity and turnover in neurons PMC

Figure 1 from The short variant of optic atrophy 1 OPA1 improves cell

Full article Mitochondrial dynamics and quality control regulate

Caspases indirectly regulate cleavage of the mitochondrial fusion

S1OPA1 overexpression exacerbated mitochondrial fragmentation and

Proteolytic Regulation of Mitochondrial Dynamics PMC

Mitochondrial Rhomboid PARL Regulates Cytochrome c Release during

神经萎缩1(OPA1)在全癌(pan-cancer)中的预后意义与生信-CSDN博客

1OPA1pancancerCSDN

Structural mechanism of mitochondrial membrane remodelling by human

Proposed mechanism of OPA1mediated mitochondrial IM fusion Human

The Role of Swelling in the Regulation of OPA1Mediated Mitochondrial

Proposed mechanism of OPA1mediated mitochondrial IM fusion Human

About Our Research Structural Cell Biology Section NIDDK

Developmental and Tumor Angiogenesis Requires the MitochondriaShaping

Mitochondrial membrane potential and oxidative stress interact to

Optic Atrophy 1 Controls Human Neuronal Development by Preventing

Mitochondrial Optic Atrophy OPA 1 Processing Is Altered in Response

The human OPA1delTTAG mutation induces adult onset and progressive

Summarizing model Schematic representation showing the different steps

A possible mechanism of how restoring LOpa1 level rescues neuronal

Diseasecausing mutations in OPA1 A Cartoon representation of the

Model of the human SOPA1 bound to membranes Lipidbinding triggers the

OMA1Mediated Mitochondrial Dynamics Balance Organellar Homeostasis

A possible mechanism of how restoring LOpa1 level rescues neuronal

OPA1 Mitochondrial

OPA1 and Mitochondria

Neuron Amine Transport

OPA1 Protein

OPA1 Immunofluorescence

OPA1 mtDNA Depletion

OPA1 Protein Domain

Neuron Ischemia-Reperfusion

OPA1 Structure

OPA1 Isoforms

OPA1 Western Blot

OPA1 Mitochondrial Fusion

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