Vps35

VPS35 and retromer dysfunction in Parkinsons disease PMC

Full article VPS35 or retromer as a potential target for

Hypothetical drug exploiting the double nature of VPS35

Parkinsons Disease Genes VPS35 and EIF4G1 Interact Genetically and

VPS35 and α-Synuclein fail to interact to modulate neurodegeneration in ...

VPS35 and Synuclein fail to interact to modulate neurodegeneration in

Parkinsons diseaseassociated VPS35 mutant reduces mitochondrial

Parkinsons diseaselinked mutations in VPS35 induce dopaminergic

The Vps35 D620N Mutation Linked to Parkinsons Disease Disrupts the

Parkinsons Disease Genes VPS35 and EIF4G1 Interact Genetically and

VPS35 and Synuclein fail to interact to modulate neurodegeneration in

PDF Mutation in VPS35 associated with Parkinsons disease impairs

Parkinsons Disease Genes VPS35 and EIF4G1 Interact Genetically and

The Vps35 D620N Mutation Linked to Parkinsons Disease Disrupts the

a VPS35 is a core component of the retromer cargorecognition complex

The impact of VPS35 D620N mutation on alternative autophagy and its

Results of the systematic literature review for VPS35 mutation

Parkinson Diseaselinked Vps35 R524W Mutation Impairs the Endosomal

VPS35 and Synuclein fail to interact to modulate neurodegeneration in

Parkinsons diseaseassociated VPS35 mutant reduces mitochondrial

Schematic representation of the A LRRK2 and B VPS35 proteins with the

(PDF) VPS35 and α-Synuclein fail to interact to modulate ...

PDF VPS35 and Synuclein fail to interact to modulate

Vps35 and the Retromer Complex Protect against Accumulation of

The impact of VPS35 D620N mutation on alternative autophagy and its

Parkinsons disease genes VPS35 and EIF4G1 interact genetically and

Parkinsons diseaseassociated VPS35 mutant reduces mitochondrial

Parkinsons Disease Causative Mutation in Vps35 Disturbs Tetherin

Parkinsons diseaseassociated VPS35 mutant reduces mitochondrial

Parkinsons Disease Genes VPS35 and EIF4G1 Interact Genetically and

Parkin mediates the ubiquitination of VPS35 and modulates retromer

The Vps35 D620N Mutation Linked to Parkinsons Disease Disrupts the

Normal VPS35 Levels and Vesicular Sorting of the Retromer Cargo CIM6PR

AAV26mediated expression of human VPS35 in the rat substantia nigra

Parkinson Diseaselinked Vps35 R524W Mutation Impairs the Endosomal

VPS35 promotes cell proliferation via EGFR recycling and enhances EGFR

a VPS35 is a core component of the retromer cargorecognition complex

Full article VPS35 or retromer as a potential target for

Table 1 from Identification of VPS35 mutations replicated in French

Figure 1 from Understanding the contributions of VPS35 and the retromer

Mitochondrial morphology and respiratory deficits in VPS35 D620N KI

VPS35 Wikiwand

Vps35 colocalizes with Amyloid Precursor Protein A Hippocampal neurons

Parkin mediates the ubiquitination of VPS35 and modulates retromer

VPS35 Deficiency Markedly Reduces the Proliferation of HEK293 Cells

Vps35 Mediates Vesicle Transport between the Mitochondria and

Mutant mice with rodspecific VPS35 deletion exhibit retinal

Subcellular VPS35 The Human Protein Atlas

VPS35 promotes cell proliferation via EGFR recycling and enhances EGFR

VPS35 expression is upregulated in breast cancer and high expression

Parkin mediates the ubiquitination of VPS35 and modulates retromer

VPS35 contributes to proliferation in PDAC cells A Western blot images

Interaction between VPS35 and RABG3f is necessary as a checkpoint to

Functional role of VPS35 in brain endothelial cells JIR

VPS35 Deficiency or Mutation Causes Dopaminergic Neuronal Loss by

Brain VPS35 overexpression reduces tau phosphorylation and pathology in

Normal retromer assembly in brain but reduced VPS35 and WASH complex in

Cellular distribution and levels of endogenous VPS35 in normal and

VPS35 Deficiency Markedly Reduces the Proliferation of HEK293 Cells

Generation of VPS35 conditional knockout mice AC Singlecell RNAseq

Conserved asymmetric VPS35 dimerization interface A Top and side

VPS35 contributes to the maintenance of genome stability A

VPS35 is overexpressed and associated with poor prognosis in GC A Venn

Mutant mice with rodspecific VPS35 deletion exhibit retinal

VPS35 David K Simon Lab

Genetic overexpression and pharmacological stabilization of VPS35

Overexpression of VPS35 in N2A-APPswe cells affects Aβ formation. a ...

Overexpression of VPS35 in N2AAPPswe cells affects A formation a

The cell surface proteome is remodelled in VPS35 KO H4 cells a Scatter

Conserved electrostatic patches mediate key VPS35 dimer interfaces

Vps35 Mediates Vesicle Transport between the Mitochondria and

VPS35 Monoclonal Antibody 8A3 APC Invitrogen MA545942

How VPS35 regulates mitochondrial translation under oxidative stress

VPS35 Monoclonal Antibody 8A3 APC Invitrogen MA545942

VPS35 Mechanisms and Pathways that Induce Pathogenesis of

VPS35 the Retromer Complex and Parkinsons Disease PMC

Frontiers VPS35Based Approach A Potential Innovative Treatment in

VPS35 the core component of the retromer complex and Parkinsons

Ibrain Vol 7 No 4

| VPS35-retromer subunit controls the main α-synuclein degradation ...

VPS35retromer subunit controls the main synuclein degradation

Frontiers VPS35Based Approach A Potential Innovative Treatment in

Genetic Basis of Parkinsons Disease Encyclopedia MDPI

Frontiers VPS35Based Approach A Potential Innovative Treatment in

Parkinsons VPS35D620N mutation induces LRRK2mediated lysosomal

Phospholipase PLA2G6 a ParkinsonismAssociated Gene Affects Vps26 and

A Mutation in VPS35 Encoding a Subunit of the Retromer Complex Causes

Figure 1 from Retromerdependent neurotransmitter receptor trafficking

UNDERSTANDING THE ROLE OF VPS 35 IN AUTOPHAGY AND IN PARKINSONS

PDF VPS35 the core component of the retromer complex and Parkinson

Parkinsons VPS35D620N mutation induces LRRK2mediated lysosomal

Formation of retromer transport carriers is disrupted by the Parkinson

Parkinsons VPS35D620N mutation induces LRRK2mediated lysosomal

Figure 5 from Impaired neurogenesis in the hippocampus of an adult

Figure 1 from Retromer Binding to FAM21 and the WASH Complex Is

Parkinsonism Related Disorders

Vacuolar protein sorting 35 Vps35 rescues locomotor deficits and

A Mutation in VPS35 Encoding a Subunit of the Retromer Complex Causes

Parkinsons VPS35D620N mutation induces LRRK2mediated lysosomal

Science China Life Sciences｜孙建伟课题组揭示VPS35/Retromer-MT1-MMP信号轴驱动黑色素瘤转移机制 ...

Science China Life SciencesVPS35RetromerMT1MMP

Figure 1 from A conserved retromer sorting motif is essential for

VPS35Retromer Multifunctional Roles in Various Biological Processes

The Structure and Function of the Retromer Protein Complex Collins

Structural Mechanism for Cargo Recognition by the Retromer Complex Cell

VPS35Retromer Multifunctional Roles in Various Biological Processes

Exploring the triad VPS35 neurogenesis and neurodegenerative

The neuronal retromer can regulate both neuronal and microglial

Vps35dependent recycling of Trem2 regulates microglial function Yin

VPS35B Colocalizes with RetromerAssociated Proteins to MVBsPVCs

The Retromer Complex From Genesis to Revelations Trends in

RetromerMediated Trafficking of Transmembrane Receptors and Transporters

Exploring the triad VPS35 neurogenesis and neurodegenerative

VPS35L localization and PM proteome effects a Immunofluorescence

PDF The neuronal retromer can regulate both neuronal and microglial

VPS35deficiency results in an impaired AMPA receptor trafficking and

Improved mammalian retromer cryoEM structures reveal a new assembly

The neuronal retromer can regulate both neuronal and microglial

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