Snx16

SNX16 and SNX2 partially colocalize in early endosome structures A431

SNX16 Regulates the Recycling of ECadherin through a Unique Mechanism

SNX16 is associated to a subset of late endosome membranes A HeLa

SNX16 localizes to Rab5positive early endosomes A Kc167 cells

SNX16 is not present on early endosomal membranes A HeLa cells

Evidence for a Role of SNX16 in Regulating Traffic between the Early

SNX16 Regulates the Recycling of ECadherin through a Unique Mechanism

Localization of SNX16 to endosomes at the Drosophila NMJ A elav C155

SNX16 activates cMyc signaling by inhibiting ubiquitinmediated

SNX16 physically interacts with Nwk A Schematic of Nwk and SNX16

Role of SNX16 in the dynamics of tubulocisternal membrane domains of

Evidence for a Role of SNX16 in Regulating Traffic between the Early

SNX16 aggravates AngIIinduced cardiac hypertrophy in mice via EGFR

SNX16 negatively regulates the migration and tumorigenesis of MCF7

SNX16 Regulates the Recycling of ECadherin through a Unique Mechanism

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

Exploring the effect of SNX16 on sepsis using bioinformatics and a

SNX16 is involved in the early stage of IAV replication cycle a

Manipulations of endosomal and BMP trafficking alter Snx16 compartments

A Nwkbinding mutant of Snx16 recapitulates the nwk phenotype A

Endogenous SNX16 localizes to the early endosome and the late

Evidence for a Role of SNX16 in Regulating Traffic between the Early

Loss of SNX16 suppresses the nwk mutant phenotype A Schematic of

Endogenous SNX16 localizes to the early endosome and the late

Working model for SNX16 function The SNX16 CC domain controls its

SNX16 promotes CRC cell proliferation by activating the cMyc signaling

SNX16 promotes tumorigenesis in mice A Subcutaneous xenograft tumor

SNX16 distribution in the absence of polymerized microtubules and

SNX16 promotes CRC cell proliferation by activating the cMyc signaling

SNX16 activates the cMyc signaling pathway by inhibiting eEF1A2

Endogenous SNX16 localizes to the early endosome and the late

SNX16 is associated to a subset of late endosome membranes A HeLa

SNX16 is overexpressed in CRC and overexpression of SNX16 is

SNX16 interacts with eEF1A2 in CRC A B The proteins pulled down by

Role of SNX16 in the Dynamics of TubuloCisternal Membrane Domains of

SNX16 regulates the migration but not proliferation of cells A

SNX16 negatively regulates tumorigenesis of MCF7 cells in vivo A

SNX16 activates cMyc signaling by inhibiting ubiquitinmediated

Analysis of SNX16 distribution by fluorescence microscopy and

mTORC1 regulates autophagosomal components recycling through SNX16

The coiledcoil domain of SNX16 is important for homooligomerization

PNAS：华中科技大学荣岳光团队发现 mTORC1 通过 SNX16 磷酸化调节自噬体成分循环 - 文献解读 - 丁香通

PNAS mTORC1 SNX16

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

SNX16 physically interacts with Nwk A Schematic of Nwk and SNX16

SNX16 Regulates the Recycling of ECadherin through a Unique Mechanism

Working model for SNX16 function The SNX16 CC domain controls its

Localization of SNX16 to endosomes at the Drosophila NMJ A elav C155

PDF SNX16 negatively regulates the migration and tumorigenesis of MCF

Overexpression of SNX16 inhibits the replication of IAV a A549 cells

SNX16 Regulates the Recycling of ECadherin through a Unique Mechanism

mTORC1 regulates autophagosomal components recycling through SNX16

Role of SNX16 in the dynamics of tubulocisternal membrane domains of

SNX16 Polyclonal Antibody Invitrogen A305721AT

mTORC1 regulates autophagosomal components recycling through SNX16

PDF Role of SNX16 in the Dynamics of TubuloCisternal Membrane

mTORC1 regulates autophagosomal components recycling through SNX16

Overexpression of SNX16 inhibits the replication of IAV a A549 cells

mTORC1 regulates autophagosomal components recycling through SNX16

R144A mutation of SNX16 attenuates its inhibitory effect on IAV

Overexpression of SNX16 inhibits the replication of IAV a A549 cells

GSEA predicts SNX16 and PAPOLG related KEEG pathways Pathways enriched

Endogenous SNX16 localizes to the early endosome and the late

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

Endogenous SNX16 localizes to the early endosome and the late

SNX16 Polyclonal Antibody PA555166

PDF SNX16 aggravates AngIIinduced cardiac hypertrophy in mice via

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

Evidence for a Role of SNX16 in Regulating Traffic between the Early

SNX16 Products

Evidence for a Role of SNX16 in Regulating Traffic between the Early

SNX16 Antibody B4 SCBT Santa Cruz Biotechnology

SNX16 Products

SNX16 distribution after glutaraldehyde fixation and after brefeldin A

SNX16 is associated to a subset of late endosome membranes A HeLa

Evidence for a Role of SNX16 in Regulating Traffic between the Early

Addgene SNX16 gRNA BRDN0001146480

Role of SNX16 in the dynamics of tubulocisternal membrane domains of

SNX16 promotes CRC cells growth in vitro A B Effects of SNX16

A Nwkbinding mutant of Snx16 recapitulates the nwk phenotype A

Role of SNX16 in the Dynamics of TubuloCisternal Membrane Domains of

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

Role of SNX16 in the Dynamics of TubuloCisternal Membrane Domains of

撤回：利用生物信息学和盲肠结扎穿刺 (CLP) 小鼠模型探索 SNX16 对脓毒症的影响,iScience - X-MOL

CLP SNX16 iScience XMOL

SNX16 negatively regulates the migration and tumorigenesis of MCF7

Role of SNX16 in the Dynamics of TubuloCisternal Membrane Domains of

SNX16 activates cMyc signaling by inhibiting ubiquitinmediated

Manipulations of endosomal and BMP trafficking alter Snx16 compartments

Loss of SNX16 suppresses the nwk mutant phenotype A Schematic of

SNX16 activates cMyc signaling by inhibiting ubiquitinmediated

SNX16 preferentially binds PI3P A diagrammatic representation of the

SNX16 activates cMyc signaling by inhibiting ubiquitinmediated

Role of SNX16 in the Dynamics of TubuloCisternal Membrane Domains of

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

SNX16 is associated to a subset of late endosome membranes A HeLa

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

Endogenous SNX16 localizes to the early endosome and the late

Loss of SNX16 suppresses the nwk mutant phenotype A Schematic of

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

Figure 1 from Evidence for a Role of SNX16 in Regulating Traffic

A Nwkbinding mutant of Snx16 recapitulates the nwk phenotype A

A Single Amino Acid Residue R144 of SNX16 Affects Its Ability to

Motility of VenusSNX16containing endosomes depends on microtubules A

Subcellular distribution of SNX16 A The subcellular distribution

Addgene SNX16PX 104216

Comprehensive analysis of GSECmiR1013pSNX16PAPOLG axis in

dSNX16 coiledcoil variants alter endosome structure localization and

dSNX16 CC variants alter endosome structure localization and

SNX16抗体 (C-11) | SCBT - Santa Cruz Biotechnology

SNX16 C11 SCBT Santa Cruz Biotechnology

A presynaptic endosomal trafficking pathway controls synaptic growth

Homology analysis of residue R144 of SNX16 The amino acid sequences of

Figure 1 from Comprehensive analysis of GSECmiR1013pSNX16PAPOLG

Figure 1 from A presynaptic endosomal trafficking pathway controls

SNX16 C11 SCBT Santa Cruz Biotechnology

HTC One M9

Human Mitochondrial DNA Haplogroup

Polyclonal Antibody

propionyl-CoA Carboxylase

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