Slc39a8

SLC39A8 gene encoding a metal ion transporter discovery and bench to

JCI Allosteric modulation of the solute carrier transporter SLC39A8

Molecular mechanisms underlying the SLC39A8 transeQTL locus A

SLC39A8 gene encoding a metal ion transporter discovery and bench to

The schizophreniaassociated variant in SLC39A8 alters protein

JCI Allosteric modulation of the solute carrier transporter SLC39A8

SLC39A8 gene encoding a metal ion transporter discovery and bench to

Loss of Slc39a8 in neurons results in Mn deficiency in the brain

The mechanism in which Slc39a8 deletion leads to hypertrabeculation and

Zinc transporter Slc39a8 is essential for cardiac ventricular

The mechanism in which Slc39a8 deletion leads to hypertrabeculation and

Schematic model of SLC39A8 A391Tinduced intestinal barrier disruption

Tissue expression of SLC39A8 Staining in kidney The Human Protein Atlas

Tissue expression of SLC39A8 Staining in rectum The Human Protein Atlas

Tissue expression of SLC39A8 Staining in placenta The Human Protein

Mn supplementation increases large Nglycans in severe SLC39A8 mutation

Tissue expression of SLC39A8 Staining in kidney The Human Protein Atlas

Tissue expression of SLC39A8 Staining in breast The Human Protein Atlas

Plasma protein Nglycan changes in severe lossoffunction SLC39A8

Tissue expression of SLC39A8 Staining in kidney The Human Protein Atlas

SLC39A8 Fusion Protein Ag14292 Proteintech

Zinc transporter Slc39a8 is essential for cardiac ventricular

Tissue expression of SLC39A8 Staining in kidney The Human Protein Atlas

PDF SLC39A8 gene encoding a metal ion transporter Discovery and

SLC39A8 was down expression and positively associated with tumor

SLC39A8 Deficiency A Disorder of Manganese Transport and Glycosylation

Positions of seven single nucleotide polymorphism in the SLC39A8 gene

Metalion transporter SLC39A8 is required for brain manganese uptake

Zinc transporter Slc39a8 is essential for cardiac ventricular

SLC39A8 Deficiency A Disorder of Manganese Transport and Glycosylation

Immunofluorescence of PRNP green SLC39A8 red LCN2 red ATP6V0A1

PDF Schizophreniaassociated SLC39A8 polymorphism is a lossof

Tissue expression of SLC39A8 Staining in liver The Human Protein Atlas

SLC39A8 allelic variants found to be associated with clinical disorders

Does SLC39A8 Ala391Thr Confer Risk of Chronic Liver Disease Anne

Tissue expression of SLC39A8 Staining in esophagus The Human

Positions of seven single nucleotide polymorphism in the SLC39A8 gene

Figure 2 from A missense variant in SLC39A8 confers risk for Crohns

MRI Findings in Individuals with Mutation in SLC39A8 MRI findings from

Slc39a8 A391T variant is associated with reduced Mn levels and impaired

SLC39A8 Expression Is Induced by Proinflammatory Stimuli in an

Tissue expression of SLC39A8 Staining in placenta The Human Protein

Generation of a Slc39a8 Hypomorph Mouse Markedly Decreased ZIP8 Zn2

Tissue expression of SLC39A8 Staining in kidney The Human Protein Atlas

Tissue expression of SLC39A8 Staining in placenta The Human Protein

JCI Insight Neuronal SLC39A8 deficiency impairs cerebellar

Metalion transporter SLC39A8 is required for brain manganese uptake

Tissue expression of SLC39A8 Staining in placenta The Human Protein

Tissue expression of SLC39A8 Staining in liver The Human Protein Atlas

The schizophreniaassociated variant in SLC39A8 alters protein

Tissue expression of SLC39A8 Staining in liver The Human Protein Atlas

A missense variant in SLC39A8 is associated with severe idiopathic

SLC39A8 Expression Is Induced by Proinflammatory Stimuli in a

Transcriptional Activation of the Human SLC39A8 Promoter by NFB

SLC39A8 predicted network linking smoking to atherosclerosis from

SLC39A8 Fusion Protein Ag14292 Proteintech

The manganese transporter SLC39A8 links alkaline ceramidase 1 to

JCI Zinc transporter Slc39a8 is essential for cardiac ventricular

Generation of the SLC39A8p393T knockin mouse Download

Evaluation of the prognostic values of solute carrier SLC family 39

SLC39A family members zincironregulated transporterlike protein

Sequence of steps that describe the first three paragraphs of the

SLC39A8Zinc Suppresses the Progression of Clear Cell Renal Cell

Frontiers SLC39A8Zinc Suppresses the Progression of Clear Cell Renal

CDD | 肿瘤内短小芽孢杆菌通过抑制NK细胞铁死亡增强肝癌抗肿瘤免疫-欧易生物

CDD NK

Figure 1 from Allosteric modulation of the solute carrier transporter

Molecular and pathophysiological aspects of metal ion uptake by the

Forced Overexpression and Knockout Analysis of SLC30A and SLC39A Family

SLC39A8p393T knockin affected dendritic spine density A Overview of

SLC39A8Zinc Suppresses the Progression of Clear Cell Renal Cell

PDF ZIP8 Member of the SoluteCarrier39 SLC39 MetalTransporter

Frontiers SLC39A8Zinc Suppresses the Progression of Clear Cell Renal

Forced Overexpression and Knockout Analysis of SLC30A and SLC39A Family

Abcam Best Science Apps Amyloid Precursor Protein Facilitates

BIGM103 SLC39A8 NM022154 Human Untagged Clone from OriGene

Frontiers SLC39A8Zinc Suppresses the Progression of Clear Cell Renal

SLC39A8mediated zinc dyshomeostasis potentiates kidney disease PNAS

Figure 1 from Molecular and pathophysiological aspects of metal ion

Relative ZIP8 mRNA levels in Slc39a8 Slc39a8 neo and

Forced Overexpression and Knockout Analysis of SLC30A and SLC39A Family

Evaluation of the prognostic values of solute carrier SLC family 39

A homologybased 3D model and structurefunction studies reveal key

Forced Overexpression and Knockout Analysis of SLC30A and SLC39A Family

Frontiers SLC39A8Zinc Suppresses the Progression of Clear Cell Renal

Clinical molecular and glycophenotype insights in SLC39A8CDG

Hepatic metal ion transporter ZIP8 regulates manganese homeostasis and

Recent Positive Selection Drives the Expansion of a Schizophrenia Risk

Molecular and pathophysiological aspects of metal ion uptake by the

SLC39A8CDG with manganese deficiency in an adult individual a case

ZIP8 Regulates Host Defense through Zinc-Mediated Inhibition of NF-κB ...

ZIP8 Regulates Host Defense through ZincMediated Inhibition of NFB

Mutations in hSLC39A8 are associated with severe Mn deficiency

ANTISLC39A8 ANTIBODY 100UL

SLC39A8Zinc Suppresses the Progression of Clear Cell Renal Cell

Forced Overexpression and Knockout Analysis of SLC30A and SLC39A Family

Plasma protein Nglycan branching following Mn supplementation in

Impact of SLC39A8p393T knockin on gene expression in the cortex and

Forced Overexpression and Knockout Analysis of SLC30A and SLC39A Family

mRNA expression of SLC39A family genes in primary tumor and

CircSLC39A8 expression was increased in OA cartilage tissues a The

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Novel homozygous variant of the SLC39A14 gene identified in the

Frontiers SLC39A8Zinc Suppresses the Progression of Clear Cell Renal

CircSLC39A8 expression was increased in OA cartilage tissues a The

In utero gene expression in the Slc39a8neoneo knockdown mouse

SLC39A8zinc inhibits the invasion and migration of ccRCC cells in

mRNA expression of SLC39A family genes in primary tumor and

Forced Overexpression and Knockout Analysis of SLC30A and SLC39A Family

Molecular Cell丨AFG3L2 和铁对 SLC25A39 的双重调控控制着线粒体谷胱甘肽的稳态 - 知乎

Molecular CellAFG3L2 SLC25A39

CircSLC39A8 expression was increased in OA cartilage tissues a The

Clinical molecular and glycophenotype insights in SLC39A8CDG

CircSLC39A8 silence increased cell viability and suppressed apoptosis

ZIP8 Regulates Host Defense through ZincMediated Inhibition of NFB

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