Sema3d

Schematic summary of Sema3D manipulation results Schematic dorsal

Sema3D does not repel MLF axons in the anterior hindbrain A Schematic

Hapln1a and Sema3d function in a common pathway to mediate Cx43

Differential Sema3d functions are transduced by signaling through the

sema3d and netrin 1b have complementary expression patterns in the

Sema3d is localized in the mesenchyme next to the CCV and in the ECs of

sema3d and netrin 1b have complementary expression patterns in the

Sema3d expression in the developing third pharyngeal pouch A Bright

AnxA2 controls the secretion of Sema3D from PDA cells allowing it to

ECspecific Sema3d acts independently of mesenchymal Sema3d and is

Sema3D influences interactions between MLF axons and hindbrain neurons

Mesenchymal Sema3d acts as a repulsive cue for CCV outgrowth AC

Schematic models of potential interactions between WntTCF and Sema3d

Sema3d expression excludes endothelium from the AV groove ab Xgal

Sema3D is involved in invasion and metastasis of PDA and is associated

Testing the Ret and Sema3d genetic interaction in mouse enteric nervous

Sema3d mRNA is expressed in NCCs and morpholino knockdown of Sema3d

Sema3d directly interacts with FLNA and affects cytoskeleton

Abundant expression of SEMA3D and PDE1A proteins in layers of

Localized Sema3D misexpression inhibits ventral RGC axons Lateral

Sema3d restrains HCC migration invasion proliferation and

The abundance of Sema3D is differentially regulated in pancreatic

Localized Sema3D misexpression inhibits ventral RGC axons Lateral

Sema3D and L11 regulate dynamic axonaxon interactions among MLF

SEMA3D Monoclonal Antibody OTI2H4 TrueMAB Invitrogen TA809917S

AnxA2 controls the secretion of Sema3D from PDA cells allowing it to

Overexpression of SEMA3D suppressed the migration in PTC cells A The

Mesenchymal Sema3d acts as a repulsive cue for CCV outgrowth AC

Morpholino mediated knockdown of hapln1a results in reduced Sema3d

The SEMA3D expression was strongly upregulated 55fold in the

Overexpression of SEMA3D suppressed the proliferation and colony

Differential Sema3d functions are transduced by signaling through the

SEMA3D recombinant protein Semaphorin3D SEMA3D Recombinant Protein

Sema3d inhibits parathyroid cell proliferation in vivo and in vitro

Sema3d GFPCreMediated Deletion of Yap and Taz Leads to Embryonic

Abundant expression of SEMA3D and PDE1A proteins in layers of

Sema3d is downregulated in HCC cell lines and tissues A Sema3d

Sema3d inhibits chemically induced EGFR signaling pathway A

The expression of SEMA3D was downregulated in PTC tissues and cell

SEMA3D contributes to PUDrelated carcinogenesis IJGM

Sema3D regulates L11 protein levels in MLF axons A B Unsaturated

Transmembrane Transporter Sema3D Serves as a Tumor Suppressor in

Sema3d Restrained Hepatocellular Carcinoma Progression Through

Sema3d directly interacts with FLNA and affects cytoskeleton

The effect of sema3E and sema3D expression in glioblastoma cells on the

Sema3d restrains HCC progression via inactivating Pi3kAkt signaling

Expression of DPT and SEMA3D genes in the human cochlea and

Overexpression of SEMA3D suppressed the migration in PTC cells A The

SEMA3D contributes to PUDrelated carcinogenesis IJGM

SEMA3D regulates the proliferation and migration of PTC through

Localization of SEMA3D mRNA and TACC2 protein in uterine endometrioid

Sema3d directly interacts with FLNA and affects cytoskeleton

Expression of DPT and SEMA3D genes in the human cochlea and

Sema3D is involved in invasion and metastasis of PDA and is associated

Overexpression of SEMA3D suppressed the proliferation and colony

Abundant expression of SEMA3D and PDE1A proteins in layers of

Sema3D regulates L11 protein levels in MLF axons A B Unsaturated

Downregulation of Sema3d promotes EMT in HCC A Correlation of FLNA

Sema3d overexpression rescues hapln1a knockdown phenotypes A

The association of Sema3D expression and clinical characteristics in

Surgical Considerations in Pulmonary Vein Anomalies Clinical Tree

Addgene Sema3dSFcHis

Semaphorin 3d and Semaphorin 3e Direct Endothelial Motility through

Semaphorin 3D promotes pancreatic ductal adenocarcinoma progression and

Distinct Compartments of the Proepicardial Organ Give Rise to Coronary

Semaphorin 3D promotes pancreatic ductal adenocarcinoma progression and

Semaphorin 3d and Semaphorin 3e Direct Endothelial Motility through

Distinct Compartments of the Proepicardial Organ Give Rise to Coronary

Sema3D Sema3F and Sema5A are expressed in overlapping and distinct

Excess L11 compensates for loss of Sema3D AC Ventral views

Sema3D Sema3F and Sema5A are expressed in overlapping and distinct

Semaphorin 3d and Semaphorin 3e Direct Endothelial Motility through

Semaphorin 3D promotes pancreatic ductal adenocarcinoma progression and

Sema3D Sema3F and Sema5A are expressed in overlapping and distinct

Semaphorin 3d and Semaphorin 3e Direct Endothelial Motility through

Distinct Compartments of the Proepicardial Organ Give Rise to Coronary

Semaphorin3D Guides Retinal Axons along the Dorsoventral Axis of the

Deficiency in the secreted protein Semaphorin3d causes abnormal

Semaphorin3D Guides Retinal Axons along the Dorsoventral Axis of the

Repulsion and Attraction of Axons by Semaphorin3D Are Mediated by

Semaphorin3D Guides Retinal Axons along the Dorsoventral Axis of the

Functional Loss of Semaphorin 3C andor Semaphorin 3D and Their

Semaphorin 3D promotes pancreatic ductal adenocarcinoma progression and

Addgene Sema3dLAPHis

Role of Semaphorin 3D in Inhibition of Colorectal Cancer Cell Migration

Sema3dNrp1 signaling regulates cellcell contacts and Actin cable

Citrullinated IGF2BP1 promotes rheumatoid synovial aggression via

Excess L11 compensates for loss of Sema3D AC Ventral views

Semaphorin 3d and Semaphorin 3e Direct Endothelial Motility through

Distinct Compartments of the Proepicardial Organ Give Rise to Coronary

Addgene Sema3dSAPHis

In silico model of how Sema3 signaling alterations regulate axon

Semaphorin 3D promotes pancreatic ductal adenocarcinoma progression and

Bone Research｜神经导向因子Sema3A软骨保护的新机制--中国科学院深圳先进技术研究院

Bone ResearchSema3A

Frontiers Transcriptome profiling of intact bowel wall reveals that

Repulsion and Attraction of Axons by Semaphorin3D Are Mediated by

In silico model of how Sema3 signaling alterations regulate axon

Transcriptome profiling of intact bowel wall reveals that PDE1A and

Role of Semaphorin 3D in Inhibition of Colorectal Cancer Cell Migration

Cx43Dependent Skeletal Phenotypes Are Mediated by Interactions between

Citrullinated IGF2BP1 promotes rheumatoid synovial aggression via

Role of Class 3 Semaphorins and Their Receptors in Tumor Growth and

Semaphorin 3D promotes pancreatic ductal adenocarcinoma progression and

Excess L11 compensates for loss of Sema3D AC Ventral views

Deficiency in the secreted protein Semaphorin3d causes abnormal

Sema3D Sema3F and Sema5A are expressed in overlapping and distinct

Immunostaining of Semaphorin 3D in colon from controls and HSCR

Sema3D L11 and Nrp1A genetically interact to promote MLF

An Unexpected Role of Semaphorin3ANeuropilin1 Signaling in Lymphatic

Semaphorin3D Regulates AxonAxon Interactions by Modulating Levels of

Model of Nrp1aSema3Dmediated guidance of left habenular axons A

Cerebral Semaphorin3D is a novel risk factor for ageassociated

Transcriptome profiling of intact bowel wall reveals that PDE1A and

Bone Research｜深圳先进院脑所杨帆团队揭示神经导向因子Sema3A软骨保护的新机制 -深港脑科学创新研究院

Bone ResearchSema3A

Semaphorin3D Guides Retinal Axons along the Dorsoventral Axis of the

Semaphorin3D Regulates AxonAxon Interactions by Modulating Levels of

Semaphorin 3D promotes pancreatic ductal adenocarcinoma progression and

RORα modulates semaphorin 3E transcription and neurovascular ...

ROR modulates semaphorin 3E transcription and neurovascular

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