Sec16a

Sec16A drives Sec body formation in INS1 cells A PCR of Sec16A and

SEC16A Variants Predispose to Chronic Pancreatitis by Impairing ERto

SEC16A is a RAB10 effector required for insulinstimulated GLUT4

Phosphorylationcoupled autoregulation of TANGO1 and Sec16A maintains

SEC16A is required for GLUT4 translocation A Quantification of PM to

Sec16A colocalizes and interacts with GRASP55 ad The cellular

Subcellular SEC16A The Human Protein Atlas

Leucinerich repeat kinase 2 regulates Sec16A at ER exit sites to allow

SEC16A is a RAB10 effector A Representative images of in situ PLA

ERES proteins Sec16A and Sec31A are recruited to the C trachomatis

ULKMediated Phosphorylation of SEC16A at S846 Is Required for Assembly

Subcellular SEC16A The Human Protein Atlas

Superresolution Imaging using STED of ERGIC53 and Sec16A in Cells

SEC16A and Intracellular Trafficking Abnormalities in Axial

Sec16A and Sec31A immunoelectron microscopy AC Localisation of

SEC16A forms a complex with PI31 and TBK1 A Immunoblot analysis

Sec16A silencing inhibits unconventional ΔF508-CFTR trafficking to the ...

Sec16A silencing inhibits unconventional F508CFTR trafficking to the

Sec16A is critical for both conventional and unconventional secretion

A GFPSec16B shows a high degree of overlap with endogenous Sec16A

Private rare deletions in SEC16A and MAMDC4 may represent novel

The ER marker SEC61BGFP does not colocalize with perinuclear SEC16A or

SEC16A is a RAB10 effector required for insulinstimulated GLUT4

Construction of pathogenic Sec16a mutation mouse model using CRISPR

Sec16A and Sec31A immunoelectron microscopy AC Localisation of

Subcellular SEC16A The Human Protein Atlas

SEC16A encircles perinuclear GLUT4 in adipocytes A and B

SEC16A antibody 200251AP Proteintech

A GFPSec16B shows a high degree of overlap with endogenous Sec16A

SEC16A Wikipedia

SEC16A Variants Predispose to Chronic Pancreatitis by Impairing ERto

SEC16A KD does not affect insulin signal transduction or TR

The organization of perinuclear RAB10 and SEC16A with GLUT4 has

SEC16A independently regulates ERtoGolgi and GLUT4 trafficking A

A GFPSec16B shows a high degree of overlap with endogenous Sec16A

SEC16A Variants Predispose to Chronic Pancreatitis by Impairing ERto

Retro2 binds directly to Sec16A a Scheme of bioorthogonal click

Increase in Sec16A detected by indirect immunofluorescence early in PV

Leucinerich repeat kinase 2 regulates Sec16A at ER exit sites to allow

Private rare deletions in SEC16A and MAMDC4 may represent novel

SEC16A Variants Predispose to Chronic Pancreatitis by Impairing ERto

$Increases in Sec16A levels in membrane and cytosolic fractions early in ...$

Increases in Sec16A levels in membrane and cytosolic fractions early in

LRRK2 colocalizes with Sec16A proteins at the dERES of cultured

A GFPSec16B shows a high degree of overlap with endogenous Sec16A

Construction of pathogenic Sec16a mutation mouse model using CRISPR

Retro2 binds directly to Sec16A a Scheme of bioorthogonal click

SEC16A Fusion Protein Ag30700 Proteintech

Phosphorylationcoupled autoregulation of TANGO1 and Sec16A maintains

Effects of Sec16 on RNF183 protein stability A Effect of Sec16A

RAB10 colocalizes with HAGLUT4GFP and SEC16A at the perinuclear

Sec16A Abnormalities Affect the Intracellular Trafficking of HLAB27 in

SEC16A antibody 294171AP Proteintech

Relocalization of Sec16A in GRASPmediated unconventional secretion of

Sec16A Abnormalities Affect the Intracellular Trafficking of HLAB27 in

SEC16A is a RAB10 effector required for insulinstimulated GLUT4

Construction of pathogenic Sec16a mutation mouse model using CRISPR

SEC16A Polyclonal Antibody Invitrogen PA552182

STED Imaging Shows Close Juxtaposition of Sec16A and ERGIC53 Remains

SEC16A Anticorps 200251AP Proteintech

Construction of pathogenic Sec16a mutation mouse model using CRISPR

Phosphorylationcoupled autoregulation of TANGO1 and Sec16A maintains

Interaction of Sec16A CCD and Sec13 A Triplicate spots of yeast

RAB10 colocalizes with HAGLUT4GFP and SEC16A at the perinuclear

Relocalization of Sec16A in GRASPmediated unconventional secretion of

Identification of p115 and Sec16A as new TRAF3 interacting proteins

TRAF3 interaction with sec16A or p115 requires its protein

PDF SEC16A is a RAB10 effector required for insulinstimulated GLUT4

Leucinerich repeat kinase 2 regulates Sec16A at ER exit sites to allow

Relocalized Sec16A colocalizes with the ER marker proteins ac

Leucinerich repeat kinase 2 regulates Sec16A at ER exit sites to allow

Subcellular SEC16A The Human Protein Atlas

SEC16A KD does not affect insulin signal transduction or TR

STED Imaging Shows Close Juxtaposition of Sec16A and ERGIC53 Remains

Relocalization of Sec16A in GRASPmediated unconventional secretion of

SEC16A forms a complex with PI31 and TBK1 A Immunoblot analysis

Relocalization of Sec16A in GRASPmediated unconventional secretion of

ULKMediated Phosphorylation of SEC16A at S846 Is Required for Assembly

Construction of pathogenic Sec16a mutation mouse model using CRISPR

Retro2 binds directly to Sec16A a Scheme of bioorthogonal click

TRAF3 interaction with sec16A or p115 requires its protein

LRRK2 colocalizes with Sec16A proteins at the dERES of cultured

STED Imaging Shows Close Juxtaposition of Sec16A and ERGIC53 Remains

Relocalization of Sec16A in GRASPmediated unconventional secretion of

Sec16Apositive structures in stressed INS1 cells are Sec bodies A

Addgene pEGFPSec16A

Structural comparison of Sec16 and Sec31 A Domain organization of

Multimodal regulation of the ERES scaffolding protein Sec16 A

Sec16A a key protein in COPII vesicle formation regulates the

MAG5SEC16A Fusion Proteins Colocalize with YFPSEC24A and SEC13AGFP

Sec16Apositive structures in stressed INS1 cells are Sec bodies A

Sec16 is a Determinant of Transitional ER Organization Current Biology

Sec16A a key protein in COPII vesicle formation regulates the

Figure 1 from Regulation of Sec16 levels and dynamics links

DYRK3 enables secretory trafficking by maintaining the liquidlike

Sec16A a key protein in COPII vesicle formation regulates the

MAG5 Encodes a Putative Ortholog with Low Sequence Similarity to Human

SEC16ASEC16 ab70722 Abcam

Requirements for the localisation of GFPSec16A in HeLa cells The top

DYRK3 enables secretory trafficking by maintaining the liquidlike

Sec16Apositive structures in stressed INS1 cells are Sec bodies A

An alternative splicing signature defines the basallike phenotype and

Punctate Structures Labeled by MAG5SEC16A Are Surrounded by the

Two Mammalian Sec16 Homologues Have Nonredundant Functions in

DYRK3 enables secretory trafficking by maintaining the liquidlike

The Noncanonical Role of ULKATG1 in ERtoGolgi Trafficking Is

COPIIASO colocalization does not require de novo COPII assembly at

Interaction and colocalization of RNF183 and Sec16A A Interaction of

Noncanonical Role of ULKATG1 in ERtoGolgi Trafficking Is Essential

Insulin promotes mobilization of HAGLUT4mEos32 from the perinuclear

Multimodal regulation of the ERES scaffolding protein Sec16 A

MAG5SEC16A Fusion Proteins Colocalize with YFPSEC24A and SEC13AGFP

An alternative splicing signature defines the basallike phenotype and

Interaction and colocalization of RNF183 and Sec16A A Interaction of

The Noncanonical Role of ULKATG1 in ERtoGolgi Trafficking Is

Interaction and colocalization of RNF183 and Sec16A A Interaction of

Sec16A a key protein in COPII vesicle formation regulates the

Sec16a Immunostaining

Sec16a SAR-1

ER Sec16a

Mammalian Cell Membrane

Kif20a Sec16a Trim 3.7

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