Ift88

Ciliary and NonCiliary Roles of IFT88 in Development and Diseases

A model for IFT88 mediated secretory pathway from ER to primary cilia

IFT88 antibody BiCell Scientific

IFT88 is required for the movement of peripheral microtubule clusters

IFT88 mutation increases Factin and nonmuscle myosin IIB expression

IFT88 Cterminal Loop Interacts with IFT144 Cleft for IFTA Entry into

IFT88 accumulations are contained within the bulge structures at the

IFT88 binds at the Cterminal end of the IFT70 superhelix A GST

A model for IFT88 mediated secretory pathway from ER to primary cilia

IFT88 accumulations are contained within the bulge structures at the

The C-terminal α-helix region of IFT88 is crucial for its interaction ...

The Cterminal helix region of IFT88 is crucial for its interaction

Different fixation methods affect IFT88 immunolabeling at cilia in

Decreased IFT88 expression with primary cilia shortening causes

IFT88 moves towards spindle poles and requires microtubules for its

Decreased IFT88 expression with primary cilia shortening causes

IFT88 accumulations are contained within the bulge structures at the

Effects of reexpression of intraflagellar transport 88 IFT88 in IFT88

IFT88 deficiency in proximal tubular cells exaggerates cisplatin

IFT88 antibody BiCell Scientific

IFT88 maintains sensory function by localising signalling proteins

Decreased IFT88 expression with primary cilia shortening causes

IFT88 deficiency in proximal tubular cells exaggerates cisplatin

Decreased IFT88 expression with primary cilia shortening causes

IFT88 influences chondrocyte actin organization and biomechanics

Pharmacological rescue of ciliary defect on IFT88 mutant cellsa

IFT88 antibody BiCell Scientific

IFT88 depletion leads to mitotic defects in HeLa cells kidney cells

IFT88 antibody BiCell Scientific

胸腺上皮细胞中 Ift88 的消耗影响老年小鼠的胸腺突触和 T 细胞分化,Anatomical Science International ...

Ift88 T Anatomical Science International

Accumulation of IFT88 and IFT140 within cilia in WDR60KO and

Knockdown of IFT88 impairs cilium function and decreases expression

Ultrastructure of Line 0 cilia and detection of IFT88 and Arl13b along

Ift88 inactivation protects the kidney from cyst development in Pkd1

Frontiers Knockdown of Ift88 in fibroblasts causes extracellular

IFT88 contributes to kfibers reincorporation into spindle after

IFT88 is required for localization of complex B proteins to transition

IFT88 deficiency in proximal tubular cells exaggerates cisplatin

IFT88 Plays a CiliaIndependent Role in Controlling OCD at Gastrulation

A model for IFT88 mediated secretory pathway from ER to primary cilia

IFT88 maintains sensory function by localising signalling proteins

SuperResolution Imaging Reveals TCTN2 DepletionInduced IFT88 Lumen

Gene therapeutic restoration of cilia morphology in Ift88 osnKO mutant

IFT88 mutations identified in individuals with nonsyndromic recessive

Ift88 is expressed in primary cilia in the organ of Cortia Schematic

Decreased IFT88 expression with primary cilia shortening causes

IFT88 antibody BiCell Scientific

Ultrastructure of Line 0 cilia and detection of IFT88 and Arl13b along

Ciliary and NonCiliary Roles of IFT88 in Development and Diseases

Localizaon of IFT88 in CEP170 KO cilia A Localizaon of IFT88 green

Representative immunofluorescence IF image of lung from an IFT88

Loss of Ift88 led to the loss of cilia and induced marked morphological

T cellexpressed Ift88 is required for proper thymocyte differentiation

Reduction of Ift88 expression in FB leads to increased ECM genes

IFT88 Plays a Cilia and PCPIndependent Role in Controlling Oriented

IFT88 maintains sensory function by localising signalling proteins

IFT88 antibody BiCell Scientific

IFT88 maintains sensory function by localising signalling proteins

Cilia protein IFT88 regulates extracellular protease activity by

A model for IFT88 mediated secretory pathway from ER to primary cilia

Loss of Ift88 does not affect Tgfβ and Wnt/β-catenin signaling but ...

Loss of Ift88 does not affect Tgf and Wntcatenin signaling but

Endothelialspecific Loss of IFT88 Promotes EndothelialtoMesenchymal

Accumulation of IFT88 within cilia in WDR34KO cells Control RPE1 cells

Decreased IFT88 expression with primary cilia shortening causes

Establishment of IFT88 KO cell lines A Integration of the donor

IFT88 Plays a CiliaIndependent Role in Controlling OCD at Gastrulation

Loss of Ift88 led to the loss of cilia and induced marked morphological

Hypomorphic expression of IFT88 leads to olfactory cilia loss and

Ciliary and NonCiliary Roles of IFT88 in Development and Diseases

IFT88 is part of a dynein1driven transport complex in mitosis a

IFT88 maintains sensory function by localising signalling proteins

The Cterminal helix region of IFT88 is crucial for its interaction

Frontiers Knockdown of Ift88 in fibroblasts causes extracellular

Negative modulation of cilia assembly and trafficking by IFT88 siRNA

IFT88 Plays a Cilia and PCPIndependent Role in Controlling Oriented

IFT88 maintains sensory function by localising signalling proteins

Ciliaassociated wound repair mediated by IFT88 in retinal pigment

Model for IFT88 function in kfibers reanchoring into mitotic

PDF The cilia protein IFT88 is required for spindle orientation in

IFT88 antibody BiCell Scientific

IFT88 controls NuMA enrichment at kfibers minusends to facilitate

Ift88 is involved in maintaining epitheliallike polarity a F and

Endothelialspecific Loss of IFT88 Promotes EndothelialtoMesenchymal

Decreased IFT88 expression with primary cilia shortening causes

IFT88 antibody BiCell Scientific

Ciliary IFT88 Protects Coordinated Adolescent Growth Plate Ossification

UFL1mediated UFMylation antagonizes IFT88 ubiquitination and

IFT88 maintains sensory function by localising signalling proteins

IFT88KAP interaction defines a conserved mechanism for kinesin2IFT

Superresolution Pattern Recognition Reveals the Architectural Map of

IFT88 a subunit of the IFTB subcomplex accumulates in Cby

Kidney function in intraflagellar transport 88 IFT88 wildtype WT

Generation of conditional intraflagellar transport 88 IFT88 knockout

Intraflagellar transport protein88 IFT88 siRNA prevents cilia

TGFβ1 – induced recruitment of human bone mesenchymal stem cells is ...

TGF1 induced recruitment of human bone mesenchymal stem cells is

Phenotype of the ift881 mutant cells a Southern blot showing that

IFT88 a subunit of the IFTB subcomplex accumulates in Cby

Functional genomic screen for modulators of ciliogenesis and cilium

Genetic reduction of cilium length by targeting intraflagellar

Administration of pharmacological agents causes changes in cilium

IFT88KAP interaction defines a conserved mechanism for kinesin2IFT

Inducible Ift88deficient mice show features consistent with mild

Molecular Medicine Reports

Loss of IFT88 IFT57 and IFT140 in rod Cep164 photoreceptors A B

Role of Ciliary Protein Intraflagellar Transport Protein 88 in the

Loss of Primary Cilia Results in the Development of Cancer in the

Ultrastructure of the ift881 flagella The flagella on ift881 mutant

A Cilia Independent Role of Ift88Polaris during Cell Migration PLOS One

Loss of IFT88 IFT57 and IFT140 in rod Cep164 photoreceptors A B

Ciliadeficient renal tubule cells are primed for injury with

Role of Ciliary Protein Intraflagellar Transport Protein 88 in the

DGKδ triggers endoplasmic reticulum release of IFT88-containing ...

DGK triggers endoplasmic reticulum release of IFT88containing

Phenotype of the ift881 mutant cells a Southern blot showing that

Genetic reduction of cilium length by targeting intraflagellar

The ciliary protein intraflagellar transport 88 is required for the

Cooperation of the IFTA complex with the IFTB complex is required for

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