Icp0

Design of a FirstinClass homoPROTAC to Induce ICP0 Degradation in

Host miR138 targets herpes simplex virus 1 HSV1 ICP0 messenger RNA

Herpes simplex virus type 1 ICP0 protein mediates activation of adeno

Cytoplasmic functions of some nonessential HSV1 proteins A ICP0

ICP0 Antagonizes ICP4Dependent Silencing of the Herpes Simplex Virus

Herpes Simplex Virus Type 1 Regulatory Protein ICP0 Aids Infection in

Relative Contributions of Herpes Simplex Virus 1 ICP0 and vhs to Loss

ICP0 colocalizes with reorganized microtubules in HSV1 infected

ICP0 is necessary for dispersal of microtubule networks in HSV1

Colocalization of DAI with ICP0 during HSV1 infection A HepG2

Nuclear retention of ICP0 in cells exposed to HDAC inhibitor or

Protein composition of herpes simplex virions in the absence of ICP0

A role for a SIM in the viral ICP0 protein in HSV1 hostpathogen

Alphaherpesvirus Proteins Related to Herpes Simplex Virus Type 1 ICP0

Herpes simplex virus type 1 ICP0 protein mediates activation of adeno

Recruitment of Herpes Simplex Virus Type 1 ImmediateEarly Protein ICP0

ICP0 translocates to the cytoplasm during the early phase of HSV1

Location of ICP4 gene ICP0 gene and adjacent genes within HSV1

Attenuated virus vaccine A The ICP0 NLS live attenuated HSV1

A PreImmediateEarly Role for Tegument ICP0 in the Proteasome

Alphaherpesvirus Proteins Related to Herpes Simplex Virus Type 1 ICP0

Translocation of ICP0 and disruption of host cell microtubules in a

Host miR138 targets herpes simplex virus 1 HSV1 ICP0 messenger RNA

Location of ICP4 gene ICP0 gene and adjacent genes within HSV1

Schematic representation of HSV1 viral constructs and ICP0 plasmids

Schematic representation of ICP0 and ICP4 isoforms observed after

Localization of SENP1 and ICP0 during infection HEp2 cells or Vero

HSV1encoded ICP0 does not reduce IRF3 protein levels in transfected

HSV-1 ICP0 Dimer Domain Adopts a Novel β-barrel Fold - PMC

HSV1 ICP0 Dimer Domain Adopts a Novel barrel Fold PMC

Design of a FirstinClass homoPROTAC to Induce ICP0 Degradation in

ICP0 and its cleavage products are rapidly degraded during HSV1

Development of an ICP0 Deletion HSV1 Virus Targeting HCC A Schematic

Herpes Simplex Virus Type 1 Regulatory Protein ICP0 Aids Infection in

ICP0 is important for HSV1 replication in neuronal cells A

Herpes Simplex Virus ImmediateEarly Protein ICP0 Is Targeted by SIAH1

PDF Cellular Localization of the Herpes Simplex Virus ICP0 Protein

Recruitment of Herpes Simplex Virus Type 1 ImmediateEarly Protein ICP0

Zhangfei inhibits ICP0 transcript and protein expression and reduces

De novo repression of HSV ICP0 gene expression is ICP4dependent A

ICP0 Antagonizes ICP4Dependent Silencing of the Herpes Simplex Virus

Nuclear retention of ICP0 in cells exposed to HDAC inhibitor or

The Herpes Simplex Virus Type 1 HSV1 Regulatory Protein ICP0

A Novel Recognition by the E3 Ubiquitin Ligase of HSV1 ICP0 Enhances

During its nuclear phase the multifunctional regulatory protein ICP0

Recruitment of Herpes Simplex Virus Type 1 ImmediateEarly Protein ICP0

What Role Does Cytoplasmic ICP0 Play in HSV1 infection Future

Localization of PML I or PML Ib and ICP0 in HSV1 infected cells A

Functional Interaction between Class II Histone Deacetylases and ICP0

Design of a FirstinClass homoPROTAC to Induce ICP0 Degradation in

ICP0 is important for HSV1 replication in neuronal cells A

Localization of Hsc70 and BAG3 during VZV and HSV infection or ICP0

ICP0 is an HSV1 tegument protein A Extracellular s17 virions were

ICP0 stimulates HSV ICP0 2GFP gene expression A Photomicrographs of

Herpes simplex virus type 1 ICP0 protein mediates activation of adeno

Herpesvirusmediated stabilization of ICP0 expression neutralizes

Herpes Simplex Virus Tegument ICP0 Is Capsid Associated and Its E3

Nuclear retention of ICP0 in cells exposed to HDAC inhibitor or

Evidence that the Herpes Simplex Virus Type 1 ICP0 Protein Does Not

Alignment of ICP0 sequences from HSV1vCPc0R and HSV1 17 The Pst

Evidence that the Herpes Simplex Virus Type 1 ICP0 Protein Does Not

Termination of Transcription of LAT Increases the Amounts of ICP0 mRNA

Herpes simplex virus 1 E3 ubiquitin ligase ICP0 protein inhibits tumor

ICP0 enables and monitors the function of D cyclins in herpes simplex

Herpesvirusmediated stabilization of ICP0 expression neutralizes

Development of an ICP0 Deletion HSV1 Virus Targeting HCC A Schematic

ICP0 is an HSV1 tegument protein A Extracellular s17 virions were

Herpes Simplex Virus Type 1 ICP0 Protein Does Not Accumulate in the

NTerminal Phosphorylation Sites of Herpes Simplex Virus 1 ICP0

Relative Contributions of Herpes Simplex Virus 1 ICP0 and vhs to Loss

Frontiers Host Intrinsic and Innate Intracellular Immunity During

Cells Free FullText HSV1 ICP0 An E3 Ubiquitin Ligase That

Herpes simplex virusinfected cell protein 0 blocks the silencing of

Infected cell protein 0 functional domains and their coordination in

Analysis of the Functions of Herpes Simplex Virus Type 1 Regulatory

Independent CisRegulatory Modules within the Herpes Simplex Virus 1

Model of nuclear HSV1 DNA sensing and inhibition by ICP0 HSV1 fusion

Characterization of HSV1 viruses that express GFPtagged ICP0 A

RCSB PDB 8UA5 Crystal Structure of infected cell protein 0 ICP0

ICP0interactome analysis A Diagram of recombinant FlagICP0

Herpes Simplex Virus Type 1 ImmediateEarly Protein ICP27 Is Required

Nuclear functions of nonessential HSV1 proteins HSV1 encodes

The Differential Requirement for CyclinDependent Kinase Activities

Schematic regulatory relationship involving HSV1 ICP0 miRH23p

The Differential Requirement for CyclinDependent Kinase Activities

Herpes Simplex Virus Type 1 ImmediateEarly Protein ICP27 Is Required

Characterization of HSV1 viruses that express GFPtagged ICP0 A

Frontiers Posttranslational modifications as a key mechanism for

PML Contributes to a Cellular Mechanism of Repression of Herpes Simplex

Stimulation of the Replication of ICP0Null Mutant Herpes Simplex Virus

Biological Analysis of HSV1 Immediateearly Proteins ICP0 ICP22 and

Independent CisRegulatory Modules within the Herpes Simplex Virus 1

Components of the RESTCoRESThistone deacetylase repressor complex are

Figure 1 from Centromere Architecture Breakdown Induced by the Viral E3

PARP1 as an Epigenetic Modulator Implications for the Regulation of

Replication of ICP0Null Mutant Herpes Simplex Virus Type 1 Is

The Differential Requirement for CyclinDependent Kinase Activities

Stages of replication compartment formation in HSV1 infected cells

Discovery of SmallMolecule Inhibitors Targeting the E3 Ubiquitin

Efficient Activation of Viral Genomes by Levels of Herpes Simplex Virus

Herpes Simplex Virus 1 HSV1 Infected Cell Protein 0 ICP0 Targets

HSV1 ICP0 An E3 Ubiquitin Ligase That Counteracts Host Intrinsic and

Suppression of HSV1 infection and viral reactivation by CRISPRCas9

Characterization of Elements Regulating the NucleartoCytoplasmic

Herpes simplex virusinfected cell protein 0 blocks the silencing of

HSV1induced N6methyladenosine reprogramming via ICP0mediated

Nuclear Sensing of Viral DNA Epigenetic Regulation of Herpes Simplex

HSV1induced N6methyladenosine reprogramming via ICP0mediated

Herpes Simplex Virus 1 HSV1 Infected Cell Protein 0 ICP0 Targets

PML Contributes to a Cellular Mechanism of Repression of Herpes Simplex

Figure 5 from The Herpes Simplex Virus Type 1 HSV1 Regulatory

Biological Analysis of HSV1 Immediateearly Proteins ICP0 ICP22 and

Characterization of Elements Regulating the NucleartoCytoplasmic

Figure 4 from A neuronspecific host microRNA targets herpes simplex

PDF Herpes Simplex Virus 1 HSV1 Infected Cell Protein 0 ICP0

Map of the HSV1 genome region expressing the LATs structure of

PDF Comparison of the Biological and Biochemical Activities of

Stimulation of the Replication of ICP0Null Mutant Herpes Simplex Virus

Efficient Activation of Viral Genomes by Levels of Herpes Simplex Virus

Fig 8 Microbiology Society

Requirements for the NuclearCytoplasmic Translocation of InfectedCell

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