As160

CalmodulinBinding Domain of AS160 Regulates Contraction but Not

Model for inhibitory effects of AS160 Nterminal PTB domain constructs

Restoration of 1433 binding in the AS160 4P mutant overcomes its

Figure 2 from The Association of ClipR59 Protein with AS160 Modulates

Glucose and insulin induce Akt and AS160 phosphorylation in MIN6B1

Figure 3 from The Association of ClipR59 Protein with AS160 Modulates

IRAP and AS160 function at different steps in GLUT4 intracellular

Figure 1 from Identification of a novel AS160 splice variant that

AS1601532 interacts with fulllength AS160 and reduces the relative

Accelerating potency of AS160 on insulinresponsive GLUT4 liberation in

AS160 phosphorylation is increased by stimuli that activate cn PKC in

The Association of ClipR59 Protein with AS160 Modulates AS160 Protein

Figure 1 from The Association of ClipR59 Protein with AS160 Modulates

Characterization of the interaction between AS160 and ClipR59 A

AS160 interacts with 1433 in an insulindependent manner A CHO

Characterization of the Role of the Rab GTPaseactivating Protein AS160

Knockdown of AS160 expression by shRNA induces basal GLUT4

Effects of in situ contraction on AS160 phosphorylation in Akt2 KO

AS160 Phosphotyrosinebinding Domain Constructs Inhibit Insulin

Characterization of the Role of the Rab GTPaseactivating Protein AS160

Akt and AS160 phosphorylation A Western blot demonstrating Akt and

AS160 Phosphotyrosinebinding Domain Constructs Inhibit Insulin

Figure 1 from Rab GTPaseActivating Protein AS160 Is a Major Downstream

AS160 knockdown is partially insulinmimetic Surface Glut4 labeling is

Emerging role of Akt substrate protein AS160 in the regulation of AQP2

AS160 Phosphotyrosinebinding Domain Constructs Inhibit Insulin

Akt and AS160 phosphorylation in control and myristoylated Neftreated

AS160 is expressed and phosphorylated in cells A AS160 mRNA

Characterization of the Role of the Rab GTPaseactivating Protein AS160

AS160 phosphorylation is increased by insulin and PDGF stimulation in

AS160 knockdown is partially insulinmimetic Surface Glut4 labeling is

Glucose and insulin induce Akt and AS160 phosphorylation in MIN6B1

Aldosterone induces AS160 expression and phosphorylation mCCD

Glucose and insulin induce Akt and AS160 phosphorylation in MIN6B1

PDF PTBdomain constructs of AS160 inhibit insulinstimulated GLUT4

Figure S6 AS160 interacts with Fsp27 and is enriched at LDCS Related

AS160 Phosphotyrosinebinding Domain Constructs Inhibit Insulin

Glucose and insulin induce Akt and AS160 phosphorylation in MIN6B1

SOLUTION Expression and phosphorylation of the as160 v2 splice variant

AS160 Regulates Insulin and Contractionstimulated Glucose Uptake in

Estrogen replacement enhances insulininduced AS160 activation and

Glucose Transport A Total GLUT4 expression B AS160

Exercise effects on γ3-AMPK activity, phosphorylation of Akt2 and AS160 ...

Exercise effects on 3AMPK activity phosphorylation of Akt2 and AS160

GSK-3β and AS160 phosphorylation. Representative immunoblot showing ...

GSK3 and AS160 phosphorylation Representative immunoblot showing

Increased AS160 phosphorylation but not TBC1D1 phosphorylation with

T4 increased AS160 phosphorylation in heart Mean SD values for A

The N terminus of AS160 that comprise the 1 1 2 region are

AS160 regulates Rab10 recruitment by insulin stimulation A

Basal and insulinstimulated phosphorylation of AS160 in skeletal

Restoring AS160 phosphorylation rescues skeletal muscle insulin

Insulin resistance after a 72h fast is associated with impaired AS160

Phosphorylation of AS160 and Its Role in GLUT4 Translocation BIOC 278

The Association of ClipR59 Protein with AS160 Modulates AS160 Protein

AS160 Modulates Aldosteronestimulated Epithelial Sodium Channel

AS160 directly associates with the Na KATPasesubunit In vitro

AS160 Modulates Aldosteronestimulated Epithelial Sodium Channel

Emerging role for AS160TBC1D4 and TBC1D1 in the regulation of GLUT4

LNK deficiency decreases obesityinduced insulin resistance by

Model for the role of the Rip11AS160 complex in insulinstimulated

PDF Exerciseinduced phosphoproteins in skeletal muscle

An illustration of Akts role in the insulin signaling pathway Akt

Mechanisms for greater insulinstimulated glucose uptake in normal and

AS160

Cooperative actions of Tbc1d1 and AS160Tbc1d4 in GLUT4trafficking

A Negative Regulatory Pathway of GLUT4 Trafficking in Adipocyte New

Frontiers AktAS160 Signaling Pathway Inhibition Impairs Infection by

Model for the involvement of PI3KAktAS160 pathway in Download

Insulinmodulated Akt subcellular localization determines Akt isoform

Mice with AS160TBC1D4Thr649Ala Knockin Mutation Are Glucose

Cooperative actions of Tbc1d1 and AS160Tbc1d4 in GLUT4trafficking

Insulin treatment results in upregulation of Akt phosphorylation sites

Akt isoformsAS160GLUT4 axis a In basal condition Akt Akt1 Akt2

Effect of insulin on subcellular translocation of GLUT4 and AS160 I

4PAS160 does not affect insulin or PDGFstimulated Akt Ser 473

Effects of Acute Muscle Contraction on the Key Molecules in Insulin and

Turning Signals On and Off GLUT4 Traffic in the InsulinSignaling

LNK deficiency decreases obesityinduced insulin resistance by

A Role for 1433 in Insulinstimulated GLUT4 Translocation through Its

AS160v2 is phosphorylated in myeloma cells and introduction of a

Figure 8 from Crystal Structures of Human TBC1D1 and TBC1D4 AS160

Insulin Stimulation of GLUT4 Exocytosis but Not Its Inhibition of

Cooperative actions of Tbc1d1 and AS160Tbc1d4 in GLUT4trafficking

A Negative Regulatory Pathway of GLUT4 Trafficking in Adipocyte New

Effect of BBR on regulating the PI3KAktAS160GLUT1 pathway Protein

Phosphorylation of Akt substrate of 160 kDa AS160 and TBC1D1 in

Frontiers Promoting Glucose Transporter4 Vesicle Trafficking along

Frontiers AktAS160 Signaling Pathway Inhibition Impairs Infection by

AS160 GLUT4 and isoformspecific skeletal muscle Akt content

The expression of AS160TBC1D4 and GLUT4 in the four chambers of mouse

Article

Phosphorylation of AS160serine 704 is not essential for exercise

Mice with AS160TBC1D4Thr649Ala Knockin Mutation Are Glucose

AS160

Expression and phosphorylation of AS160 A Representative immunoblot

Insulin Signaling Pathway Antibodiescom

PDF Increased Phosphorylation of Akt Substrate of 160 kDa AS160 in

A Method to Identify Serine Kinase Substrates Journal of Biological

Metformin promoted OxLDLimpaired phosphorylation of AktAS160 pathway

Heat treatment increases insulinstimulated glucose uptake and

Insulin signaling in skeletal muscle ADP adenosine diphosphate Akt

SWELL1 protein expression regulates insulinstimulated PI3KAKT2AS160

Phosphorylation of AS160serine 704 is not essential for exercise

Glucose infusion causes insulin resistance in skeletal muscle of rats

General scheme of insulin signaling in adipocytes with the main

Phosphorylation of AS160serine 704 is not essential for exercise

Figure 21 from Mechanisms for the sustained effects of prior exercise

AS1601532 interacts with fulllength AS160 forms heterodimers and

AktAS160 Signaling Pathway Inhibition Impairs Infection by Decreasing

Activation of the glucose receptor Glut4 by insulin BioRender

Activation of InsrAktAS160 signaling in both differentiated mouse

Electrical stimulation ES induces ATPdependent AktAS160

Effect of insulin on phosphorylation of AS160 After incubation of

AktAS160 Signaling Pathway Inhibition Impairs Infection by Decreasing

TNF action on AS160 IR Akt substrates and tyrosine Download

Effect of BBR on regulating the PI3KAktAS160GLUT1 pathway Protein

Rab8aAS160MSS4 Regulatory Circuit Controls Lipid Droplet Fusion and

Basal AMPK ACC AS160 and Akt AMPK subunit ACC AS160 and Akt

Insulin and exercise signalling in skeletal muscle control pathways

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